To determine whether the prolonged allodynia, spontaneous pain, and depression-like behavior observed in T cell-deficient mice result from more pronounced or prolonged (neuro)inflammation, we measured mRNA expression of the prototypical pro-inflammatory cytokines Il1b and Tnf at the site of CFA injection (paw), and in the CNS (ipsilateral lumbar spinal cord (SC) and contralateral prefrontal cortex (PFC)). The gene discussed is IL1B; the disease is depressive disorder.