Myasthenia gravis (MG) is an autoimmune disease that is primarily mediated by antibodies against the acetylcholine receptor (AChR) on the postsynaptic membrane of the neuromuscular junction (NMJ) and muscle-specific tyrosine kinase (MuSK), which leads to postsynaptic membrane transmission dysfunction involving both cellular immunity and complement (Gilhus and Verschuuren, 2015; Gilhus, 2016). Here, MUSK is linked to myasthenia gravis.