In addition to priming CD8+ T cell responses within tumor‐draining lymph nodes, the cDC1 subset of DC also regulates effector CTL trafficking to tumors, as those DC that remain within tumor tissue can secrete CXCL9/CXCL10 chemokines to attract CXCR3+ effector CTL,11, 12 a process relevant to both endogenous antitumor T cell responses as well as therapeutic regimens that rely on administration of exogenously activated CTL. This evidence concerns the gene CXCR3 and neoplasm.