In experimental autoimmune encephalomyelitis, an MS mouse model, enhanced expression of the miR‐183 cluster in Th17‐lymphocytes increased the lymphocytes’ production of pathogenic cytokines and autoimmunity through the repression of FOXO1. 49While this link requires confirmation in human samples, hsa‐miR‐182‐5p was found to be upregulated in the whole blood of paediatric multiple sclerosis patients.51 The gene discussed is FOXO1; the disease is experimental autoimmune encephalomyelitis.