This activity was particularly relevant in cancer biology since the mRNA stability of well-known oncogenes like B-cell 2 (Bcl2) and Serine/arginine-rich splicing factor 3 (SRSF3), as well as the tumor suppressor protein 53 (p53), were demonstrated to be regulated by hnRNPL [16,17,18]. The gene discussed is SRSF3; the disease is cancer.