CDKN2D and posterior cortical atrophy: Moreover, silencing of GPR107 in AI-PCa cells decreased the expression levels of MKI67, a well-known proliferation marker associated to biochemical recurrence in PCa [54], and, in PC-3 cells, tended to increase the expression of CDKN2D, a cell-cycle inhibitor involved in the growth arrest at senescence of PCa cells [55].