EZH2 and posterior cortical atrophy: Interestingly, we also found that the decrease in the aggressiveness of PCa cells in response to GPR107-silencing could also involve a diminished expression of the splicing variants SST5TMD4, In1-ghrelin and AR-v7, as well as EZH2, four elements previously reported as key oncogenic factors in PCa and/or main drivers of CRPC [11,30,58,59].