CD36 and graft versus host disease: These include pathways regulating phagocytosis of fungal pathogens in the context of diminished expression of fungal PRRs CD36, MRC1, and complement receptor 3; induction of AIM2, IFI16, and pyrin inflammasomes; and up-regulation of mediators of chronic inflammation, which could be biomarkers for allograft dysfunction, rejection, and GVHD in transplantation.