It is worth mentioning other genes highly differentially expressed in the LCM study even if not directly functionally associated with myelination: GGCX, and MOCS. GGCX (Gamma-Glutamyl Carboxylase) was upregulated in MSA patients, and it is essential for activating vitamin K-dependent proteins [69]. Here, GGCX is linked to multiple system atrophy.