In primary tumors, PR-A expression showed negative correlation with miR-92a-3p and positive correlation with miR-26b-5p, but the strength of this correlation was moderate, as expected because of variable stromal content in resected tumor specimens, the likely influence of variability in the range of plasma progesterone levels in postmenopausal women and differences in expression of a compliment of coregulators and downstream effectors. This evidence concerns the gene S100A6 and neoplasm.