In this context, UCA1 upregulation promotes a competing situation for miR-495 and miR-184 with NRF2 [117] and ‘oxidative stress induced growth inhibitor 1′ (OSGIN1) [96], respectively, to upregulate their expression and promote a better survival to drug treatments of NSCLC cells [117] and hepatocellular carcinoma (HCC) cells [96]. This evidence concerns the gene OSGIN1 and hepatocellular carcinoma.