Th2 cytokines are responsible for the regular management of an allergic inflammatory cascade that occurs in asthma, such as changing the class of secretory antibodies from plasma cells to IgE (IL-13 and IL-4), the differentiation of mast cells (IL-3 and IL-4 and IL-13), maturation and survival of eosinophils (IL-3, IL-5 and GM-CSF) and summons of basophils (IL-3 and GMCSF) (11–13). This evidence concerns the gene IL3 and asthma.