Inspired by the microarray data and the previous studies [6, 25], we then detected the expression of several key glycolytic genes in control, SALL4 knockdown, and SALL4-overexpressing gastric cancer cells, including HK-2, lactate dehydrogenase A (LDHA), phosphofructokinase (PFKL), phosphoglycerate kinase 1 (PGK1), Glucose transporter type 1 (GLUT1), and pyruvate kinase M2 (PKM2). The gene discussed is PFKL; the disease is gastric cancer.