Of the 45 mitochondrial termini altered during infection, 21 (46.7%) were also altered in chemical apoptosis (Fig. 6C), including T3S-dependent neo-N termini in three proteins involved in apoptosis, namely, endonuclease G (MTS removal), 39S ribosomal protein S30 (P1’ = Ala26; i.e., proteolysis to the N terminus of Ala26), and histidine triad nucleotide-binding protein 2 (P1’ = Ala31) (Table 3). This evidence concerns the gene HINT2 and infection.