In addition, MALAT1 forms a complex with the serine/arginine-rich splicing factor 1 (SRSF1), the inhibitor of the DNA binding 4, HLH protein (ID4), and mutant p53, and regulates the alternative splicing of the vascular endothelial growth factor A (VEGFA) mRNA precursor (pre-mRNA), which increases the angiogenic potential of breast cancer cells [80]. This evidence concerns the gene SRSF1 and breast carcinoma.