Subjects rehospitalized during the observation (due to both cardiac and non-cardiac disorders) had significantly higher concentration of Gal-3 at baseline in an acute phase of MI vs. those who were not hospitalized during follow up (13.7 ± 7.3 ng/mL vs. 8.5 ± 4.5 ng/mL, p = 0.002; 15.9 ± 8.1 ng/mL vs. 10.9 ± 4.9 ng/mL, p = 0.02, respectively). Here, LGALS3 is linked to myocardial infarction.