FCGR2A and Guillain-Barre syndrome: As phagocytosis, cellular cytotoxicity, cytokine production, and other immune responses depend on efficient FcγR‐IgG interactions, the higher frequency of FcγRIIIa‐F/F158 among patients with the mild form of GBS may indicate this genotype reduces the affinity of IgG binding and in turn impairs immune complex clearance and decreases subsequent inflammation.13, 35, 36