PPAT and posterior cortical atrophy: Based upon these observations (see Table S1), one could speculate that the lower levels of PUFA in PPAT, in PPAT in men with PCa, and the malignant prostate gland itself are due to these PUFAs being released and converted into prostaglandins, leukotrienes, and related compounds, which was also suggested by Careaga and colleagues [54].