CIN tumors can be recognized by the accumulation of mutations in specific oncogenes, i.e., KRAS protooncogene GTPase (KRAS) and B-Raf protooncogene serine/threonine kinase (BRAF), and tumor suppressor genes, such as adenomatous polyposis coli (APC) and tumor protein p53 (TP53) [9,10,11]. Here, TP53 is linked to cervical squamous intraepithelial neoplasia.