An increase in plasma glucagon concentration also causes free fatty acid oxidation and ketone production.[14] These possible mechanisms in combination with the use of SGLT-2 inhibitors can make diabetic patients more prone to develop eu-DKA.[8,14] In our patient, urinary tract infection, dietary restriction, and fasting for therapeutic purposes, as well as non-utilization of insulin were thought to be the predisposing factors for the development of eu-DKA. Here, GCG is linked to urinary tract infection.