CAV1 and Hepatic fibrosis: We have recently reported that BRG1 activates the transcription of caveolin-1 (CAV1) in endothelial cells, which in turn represses eNOS activity thus limiting the bioavailability of NO in the liver; deficiency of BRG1 rescues eNOS activity and protects from thioacetamide (TAA) induced liver fibrosis (Shao et al., 2020).