NF-κB was evaluated because the TLR3/4-dependent regulation of NF-κB is potentiated when WDFY1 is over-expressed (Hu et al., 2015) and NF-κB is a core neuroinflammatory molecule involved in the pathogenesis of schizophrenia (Dwir et al., 2019). This evidence concerns the gene WDFY1 and schizophrenia.