Here, we propose that IFN‐γ, released from DCs engulfing autologous apoptotic neutrophils, has the potential to modulate IL‐17 signalling and thus contribute to the maintenance of a steady‐state condition, or it is involved in supporting physiological autoimmunity, which upon external insults may modify the balance of the immunological network leading to an altered steady state contributing to the development of a pathological outcome. Here, IL17A is linked to Autoimmunity.