Thus, a molecule which exhibits selectivity towards normal and cancer breast cells could find a potential application as a strategy to lower the Nrf2 level and improve current breast cancer therapies, or as could be proposed for SFN based on the literature data, as a strategy to increase the detoxification system of normal cells with no effect on cancer tissue, which in turn could mitigate the toxic effects of therapeutics. The gene discussed is NFE2L2; the disease is cancer.