Indeed, in the Copenhagen General Population study with 46,200 individuals, the risk of myocardial infarction was the highest in FH patients with Lp(a) > 50 mg/dL (105 nmol/L) [hazard ratio (HR) = 5.3, 95% CI 3.6–7.6], followed by FH and Lp(a) values ≤50 mg/dL (≤105 nmol/L) (HR = 3.2, 95% CI 2.5–4.1) compared with the reference group of subjects without FH and Lp(a) values ≤50 mg/dL (≤105 nmol/L) [41]. Here, LPA is linked to familial hyperaldosteronism.