MiR‐20b acts as a tumor suppressor in the development of thymoma by repression of NFAT5 and CAMTA1 expression to inhibit NFAT signaling.24 MiR‐7 can control CCL21 release, which is essential for thymoma germinal center development.25 The differentially expressed miRNAs are also presented with KITLG expression in a heatmap (Fig 4b). This evidence concerns the gene CCL21 and neoplasm.