In addition to platelet number, the severity of COVID‐19 disease progression also correlates closely with levels of the pro‐inflammatory mediators such as CXCL10 (IFN‐inducible protein 10) and TNFα, both associated with the pathology and progression associated with hyperinflammatory condition associated with ARDS (Bautista et al., 2013; Tang et al., 2020; Yang et al., 2020). This evidence concerns the gene TNF and COVID-19.