BDNF and Parkinson disease: Present investigation revealed that TG infection might repair cataleptic, asymmetric, and pain behaviors in the PD control rats by the following ways: (1) enhancing the release of dopamine in the striatum, (2) inhibiting the NMDA-selective glutamate receptor within the striatum by increasing the synthesis of kynurenic acid and improving the striatal BDNF level of the rescued neurons against 6-OHDA, and (3) enhancing the TAC levels and decreasing the MDA levels.