Our results showed that Ms (pMV-1768) infection inhibited serum TNF-α production (****p < 0.01, Figure 9B) and promoted bacterial survival compared to the Ms (pMV) control group, as illustrated by the increased CFUs in different organs, including the liver (*p < 0.001, Figure 9C), spleen (****p < 0.0001, Figure 9D), and lungs in the WT BMDM adoptive transfer mice (**p < 0.01, Figure 9E). Here, TNF is linked to infection.