In the light of growing evidence that cardiac defects in patients (also with partial deficiency of laminin) could be underreported in literature (Nguyen et al., 2019), it is possible that histological features of cardiomyopathy may be manifested in hearts from older dy2J/dy2J animals, despite no clear changes in electrophysiological parameters. The gene discussed is LAMB2; the disease is cardiomyopathy.