Therefore, we further assessed the inhibition of cell viability of a spectrum of chemoresistant lung cancer cells (H460, R-H460, and A549 cells) treated with conventional chemotherapeutic drugs that are well-known inducers of apoptosis, including etoposide (DNA-damaging agent), gefitinib (EGFR inhibitor), and Taxol (microtubule inhibitor), for 24 h22. Here, EGFR is linked to lung carcinoma.