The pathogenesis of PCOS is poorly understood, but chronic infections like those that characterise this disease are associated with an increase in oxidative stress and systemic inflammation, and in lipid peroxidation markers, myeloperoxidase, c-reactive protein (CRP), inflammatory cytokines, adhesion molecules, and blood lymphocytes and monocytes [13,14,15]. This evidence concerns the gene MPO and polycystic ovary syndrome.