IL17A and idiopathic pulmonary fibrosis: Dysregulated activation of Wnt/β-catenin signaling, inflammasomes, CC chemokine ligand 18 (CCL18), M1/M2 macrophage polarization, IL-17, IL-6, IL-1β, and transforming growth factor-beta (TGF-β) are immune cells and mediators involved in IPF [79].