STAT3 and cervical carcinoma: Its overexpression showed a significant suppression of cell viability, growth and migration in the human cervical cancer cell line C33A cells, via suppression of the Janus kinase (JAK)1/ Signal transducer and activator of transcription (STAT)3 pathway and the downstream factors of STAT3, such as cyclin D, Bax, VEGF and matrix metalloproteinase (MMP)2.