Its overexpression showed a significant suppression of cell viability, growth and migration in the human cervical cancer cell line C33A cells, via suppression of the Janus kinase (JAK)1/ Signal transducer and activator of transcription (STAT)3 pathway and the downstream factors of STAT3, such as cyclin D, Bax, VEGF and matrix metalloproteinase (MMP)2. Here, VEGFA is linked to cervical cancer.