PCa is typically associated with overexpression of miRNAs (e.g., miR-21, miR-32, miR-221, miR-222, miR-181, miR-18a, and miR-429) that have a crucial function in the regulation of PI3K/AKT, the epithelial-to-mesenchymal transition (EMT), cell proliferation, apoptosis, and androgen receptor (AR) expression [65, 66]. This evidence concerns the gene AKT1 and posterior cortical atrophy.