ANO5 and autosomal recessive limb-girdle muscular dystrophy type 2L: Several muscular dystrophy models, such as syntrophin α1 null mice and murine models for LGMDR12 (LGMD2L) and LGMDR1, showed aberrant muscle regeneration with longstanding necrosis and impaired exercise and contractile properties with aberrant neuromuscular junctions [47–49].