In breast cancer, a previous study showed that MSI2 helped to maintain the epithelial state and repressed breast cancer cell EMT and that MSI2 overexpression induced the expression of the epithelial marker E-cadherin and reduced that of the mesenchymal marker SLUG, whereas knockdown of MSI2 expression in breast cancer cells promoted loss of epithelial identity [14]. Here, SNAI2 is linked to breast cancer.