KRAS and lung adenocarcinoma: Lastly, in a very recent work by LeBoeuf et al., KRAS-driven murine lung adenocarcinoma cells with LOF mutations of KEAP1 displayed an enhanced antioxidant capacity and an altered metabolism, ultimately becoming highly dependent on the exogenous uptake of non-essential aminoacids (NEAAs) such as asparagine, glycine and serine, both in vitro and in vivo.