All clinico-pathological features that differed significantly between patients with (s-FL) or without (nt-FL) HT, including Ann Arbor stage, FLIPI score, LDH elevation, and bone marrow involvement (Table 1), and additional clinically relevant features, including sex, age, and FL grade were assessed for correlation with aldolase A and GAPDH expression levels. The gene discussed is GAPDH; the disease is hematocrit.