Likewise, in the in vitro angiogenesis assay, OmAd-CM silencing CXCL2 also significantly suppressed the OmAd-CM-induced capacity of GC cells to induce EC recruitment (Fig. 2g, h) and tube formation (Fig. 2i, j) compared with control and siNT OmAd-CM in both GC cells. Here, CXCL2 is linked to gastric cancer.