Further, integrin β3, previously implicated in the development and progression of AML [34], was more highly expressed on BCR-ABL1T315I+ versus BCR-ABL1+ or empty vector-transduced BA/F3 cells by immunoblotting (Figs. 3a and S4C) and super-resolution microscopy [35] (P = 0.0228, Figs. 3b and S4D). The gene discussed is BCR; the disease is acute myeloid leukemia.