High SNHG16 not only contributes to the promotion of HCC proliferation through the miR‐302a‐3p/FGF19 axis, but also sponges miR‐4500 and targets STAT3, and sponges miR‐195 to aggravate the tumorigenesis and development of HCC 29‐31. Here, FGF19 is linked to hepatocellular carcinoma.