Turnwald and colleagues (2019) recently found that telling individuals that they were at high vs. low genetic risk for obesity (via either cardiorespiratory exercise capacity or physiological satiety) impacted not only their subjective ratings of exercise and fullness, but also their actual exercise and satiation-related physiology (i.e., metabolic gas exchange, ventilatory flow rate, and glucagon-like peptide-1 response). Here, GCG is linked to Obesity.