ITGAE and Psoriasiform dermatitis: While CD103 has reportedly no obvious pathogenic role in psoriasiform skin lesions of transgenic (Tg) mice overexpressing human transforming growth factor (TGF)-β1 under the regulation of the keratin 5 promoter within the epidermis (K5.hTGFβ1 Tg mice), which displayed a spontaneous development of psoriatic dermatitis29, the potential role of CD103 in the control of the pathogenesis of psoriasiform dermatitis and other cutaneous inflammatory disorders remains elusive.