This enrichment was expected because these pathways are involved in infection like Vascular endothelial growth factor (VEGF) pathway which facilitates interaction between pathogens and host cells [53], the mitogen-activated protein kinase (MAPK) which is important in modulating host and pathogen interactions [54], epidermal growth factor receptor (EGFR) which modulates cell survival, proliferation, differentiation, and migration/invasion [55], and Peroxisome proliferator-activated receptor (PPAR) which gets activated during bacterial infection [53]. This evidence concerns the gene WNK2 and infection.