L1CAM and Alzheimer disease: These pathways were mostly involved in biological processes such as immune system responses (e.g., MHC class II antigen presentation), mitochondrial function (e.g., TCA cycle and respiratory electron transport), neurogenesis, synaptic function, and neurotransmitter signaling (e.g., L1CAM interactions, GABA receptor activation, neurotransmitter receptors and postsynaptic signal transmission, and transmission across chemical synapses pathways) that have been implicated in AD pathogenesis [87,97,98,99,100,101,102,103].