Although the role of KIM-1 in AKI has been usually viewed as anti-inflammatory (as a receptor to phosphatidylserine, it increases the uptake of apoptotic and necrotic bodies) and involved in tubular cells’ repair [15], Tian et al. [26] showed that KIM-1 plays an important role in macrophage migration to injured tubular cells in AKI, and the mitogen-activated protein kinase signaling pathway may be involved in this process. Here, HAVCR1 is linked to acute kidney injury.