The assessment of native and modified LL37 expression in tissues and circulation can help to clarify the characteristics and the outcomes of inflammation in infectious diseases, as well as in those chronic diseases in which the LL37 is up-regulated, as in psoriasis, as well as in other diseases such as RA, in which the citrullination of autoantigens appears to play a prominent pathogenic role [6,7,8,9,10,11,12]. Here, CAMP is linked to psoriasis.