Previous studies show that AKIP1 can be retained inside the nuclei through interaction with nuclear factor‐κB (NF‐κB), thus activating the NF‐κB‐dependent transcription.7, 8 Elevated AKIP1 expression occurs in both tumour cells and in the plasma of patients diagnosed with many types of cancers.9 The gene discussed is AKIP1; the disease is neoplasm.