In addition, conditional analyses of the HAPLN4 locus for schizophrenia suggested that this association was primarily explained by another gene, GATAD2A. Interestingly, GATAD2A did not survive correction in the BIP TWAS (P = 3.8 × 10−3), and implicated HAPLN4, in contrast to schizophrenia. The gene discussed is GATAD2A; the disease is schizophrenia.