PAHSAs received considerable attention because their levels were significantly decreased in serum of insulin-resistant patients [4], breast cancer patients [5], and the milk of obese women compared to lean mothers [6], as well as suppressing inflammatory markers [7], decreasing T-cell activation in ulcerative colitis [8], and improving glucose tolerance by stimulating insulin secretion via GPR120 signaling [1]. The gene discussed is INS; the disease is breast carcinoma.